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Just Get More Fiber!
- Thread starter Costas
- Start date
- Replies 26
- Views 2K
Διαπίστωσαν ότι ένα στέλεχος βακτηρίων, που ονομάζεται Lawsonibacter asaccharolyticus (L. asaccharolyticus), συνδέθηκε έντονα με την κατανάλωση καφέ.
https://www.oloygeia.gr/diatrofi/poso-kafe-gia-kala-vaktiria-entero/
Coffee consumption is associated with intestinal Lawsonibacter asaccharolyticus abundance and prevalence across multiple cohorts.
https://www.nature.com/articles/s41564-024-01858-9
https://www.oloygeia.gr/diatrofi/poso-kafe-gia-kala-vaktiria-entero/
Coffee consumption is associated with intestinal Lawsonibacter asaccharolyticus abundance and prevalence across multiple cohorts.
https://www.nature.com/articles/s41564-024-01858-9
Fennel Seeds
Chewing fennel seeds is considered beneficial for gut health, have been traditionally used in various cultures for their digestive properties. They may help alleviate bloating, gas, and indigestion. The seeds contain a compound called anethole that can relax the muscles in the gastrointestinal tract, which may aid in digestion. Additionally, fennel seeds have antioxidant and anti-inflammatory properties, which can contribute to overall gut health.
use : chew (5 to 10 minutes) the seeds before or after meals.
tested&working
reference :
CCF Tea
Chewing fennel seeds is considered beneficial for gut health, have been traditionally used in various cultures for their digestive properties. They may help alleviate bloating, gas, and indigestion. The seeds contain a compound called anethole that can relax the muscles in the gastrointestinal tract, which may aid in digestion. Additionally, fennel seeds have antioxidant and anti-inflammatory properties, which can contribute to overall gut health.
use : chew (5 to 10 minutes) the seeds before or after meals.
tested&working
reference :
CCF Tea
(2025) Clinical trial - novel nutritional formula treats gut microbial overgrowth
https://medicalxpress.com/news/2025-04-clinical-trial-nutritional-formula-effectively.html [2]
https://mbiota.com/
Mastiha Oil for Crohn Disease, IBS
https://www.pipiscrew.com/threads/fresh-oregano-tea-benefits.151309/post-150418
https://medicalxpress.com/news/2025-04-clinical-trial-nutritional-formula-effectively.html [2]
https://mbiota.com/
Mastiha Oil for Crohn Disease, IBS
https://www.pipiscrew.com/threads/fresh-oregano-tea-benefits.151309/post-150418
metabolic endotoxemia
= the lipopolysaccharides (aka LPS or else Endotoxins) found in the bacteria cell wall, that (gut) bacteria generate, fall in blood stream (due leaky gut).
sdLDL = doing the atherosclerosis. sdLDL easily oxidized. Going into endothelium of blood vessels and made the atherosclerosis.
the lipopolysaccharides bind to LDL and make it sdLDL (here is possible your LDL count to be OK, but sdLDL not)
sdLDL associated with fatty liver which associated with gut problems.
Stress increasing the lipopolysaccharides to your bloodstream ( description above^ ) even when you dont have gut problems, for moment/hours can make it leaky gut. Here is how @ the brain communicates via vagal nerve with the gut and orders to release cortisol hormone and this releases also peptides from mastcells then the gut lining destroyed (inflammation & permeability).
Eating junk food also helps lipopolysaccharides penetrate the gut.
Bad bacteria lipopolysaccharides penetrate the gut.
70% of immune system is in the gut, T cells and B cells exist there, LPS when passing to bloodstream stimulate those cells and resulting to produce inflammatory cytokines molecules (inflammation), cause immune problem as well.
Inflammatory cytokines molecules travel to the brain, passing the BBB and causes inflammation in the brain (mental fog / depression). Depression associated with atherosclerosis.
LSP goes to the liver and cause fatty liver [2] and the muscles made insulin resistant. You get a metabolic syndrome,
All those markers are of atherosclerosis. The metabolic syndrome causes inflammatory fat, the LPS going into the healthy fat tissues, activates again the immune system, that's why we found inflammatory cells in the fat (obesity person : almost the 50% of the fat are inflammatory molecules).
source (Dr. Pradip Jamnadas)
ref - 2024 - Metabolic Endotoxemia - From the Gut to Neurodegeneration
(JUL/2025) imidazole propionate molecule produced by gut bacteria causes atherosclerosis
GPT :
LDL-C (Low-Density Lipoprotein Cholesterol) : Refers to the total cholesterol content within all LDL particles in the bloodstream. It is a common measure used in lipid panels to assess cardiovascular risk. Includes all sizes of LDL particles, both large and small, and is typically measured in milligrams per deciliter (mg/dL).
sdLDL (Small Dense LDL) : Refers to a subclass of LDL particles that are smaller and denser than typical LDL particles. These particles are often considered more atherogenic (more likely to promote the formation of fatty plaques in arteries) than larger, less dense LDL particles. Elevated levels of sdLDL are associated with a higher risk of cardiovascular events, even when total LDL-C levels are within normal ranges.
= the lipopolysaccharides (aka LPS or else Endotoxins) found in the bacteria cell wall, that (gut) bacteria generate, fall in blood stream (due leaky gut).
sdLDL = doing the atherosclerosis. sdLDL easily oxidized. Going into endothelium of blood vessels and made the atherosclerosis.
the lipopolysaccharides bind to LDL and make it sdLDL (here is possible your LDL count to be OK, but sdLDL not)
sdLDL associated with fatty liver which associated with gut problems.
Stress increasing the lipopolysaccharides to your bloodstream ( description above^ ) even when you dont have gut problems, for moment/hours can make it leaky gut. Here is how @ the brain communicates via vagal nerve with the gut and orders to release cortisol hormone and this releases also peptides from mastcells then the gut lining destroyed (inflammation & permeability).
Eating junk food also helps lipopolysaccharides penetrate the gut.
Bad bacteria lipopolysaccharides penetrate the gut.
70% of immune system is in the gut, T cells and B cells exist there, LPS when passing to bloodstream stimulate those cells and resulting to produce inflammatory cytokines molecules (inflammation), cause immune problem as well.
Inflammatory cytokines molecules travel to the brain, passing the BBB and causes inflammation in the brain (mental fog / depression). Depression associated with atherosclerosis.
LSP goes to the liver and cause fatty liver [2] and the muscles made insulin resistant. You get a metabolic syndrome,
- blood sugar goes up
- triglycerides go up
- HDL goes down
All those markers are of atherosclerosis. The metabolic syndrome causes inflammatory fat, the LPS going into the healthy fat tissues, activates again the immune system, that's why we found inflammatory cells in the fat (obesity person : almost the 50% of the fat are inflammatory molecules).
source (Dr. Pradip Jamnadas)
ref - 2024 - Metabolic Endotoxemia - From the Gut to Neurodegeneration
sdLDL to be a better predictor of cardiovascular (CV) risk than LDL-C, even in patients with low cholesterol (LDL-C < 100mg/dL).
Similarly, the JUPITER trial linked sdLDL to CV deaths . . . even in patients with very low LDL-C. source
(JUL/2025) imidazole propionate molecule produced by gut bacteria causes atherosclerosis
GPT :
LDL-C (Low-Density Lipoprotein Cholesterol) : Refers to the total cholesterol content within all LDL particles in the bloodstream. It is a common measure used in lipid panels to assess cardiovascular risk. Includes all sizes of LDL particles, both large and small, and is typically measured in milligrams per deciliter (mg/dL).
sdLDL (Small Dense LDL) : Refers to a subclass of LDL particles that are smaller and denser than typical LDL particles. These particles are often considered more atherogenic (more likely to promote the formation of fatty plaques in arteries) than larger, less dense LDL particles. Elevated levels of sdLDL are associated with a higher risk of cardiovascular events, even when total LDL-C levels are within normal ranges.
| Examination | 22/JAN | 05/MAY | Range |
|---|---|---|---|
| Ουρία (Urea) | 47 mg/dl | 27 mg/dl | 10 - 50 |
| Κρεατινίνη (Creatinine) | 1.15 mg/dl | 1.08 mg/dl | 0.6 - 1.3 |
| Σίδηρος (Iron) | 65.2 μg/dl | 88.8 μg/dl | 33 - 193 |
| Φερριτίνη (Ferritin) | 125.0 ng/ml | 163 ng/ml | 30 - 400 |
| Παραθορμόνη (Parathormone) | 35.8 pg/ml | 35.9 pg/ml | 15 -65 |
| Ινσουλίνη (Insulin) | 2.64 μU/ml | 2.79 μU/ml | 2.6 - 24.9 |
| Vitamin B12 | 264.0 pg/ml | 620 pg/ml | 197-771 |
| Φυλλικό οξύ (Folic acid) | 4.0 ng/ml | 15.4 ng/ml | 3.89 - 26.8 |
| Vitamin D (OH form) | 43.30 ng/ml | 93.6 ng/ml | 30 - 100 |
03/OCT - appointment to holistic, suggested for 3 months
| Supplement | as | Dose | RDA |
|---|---|---|---|
| Solgar Folate 400mcg | Metafolin | 1 pill at breakfast | 400mcg |
| Solgar B-12 Nuggets 1000mcg | Methylcobalamin | 1 nugget at breakfast | 2.4mcg |
| Now Foods Vitamin D-3 10000IU | Cholecalciferol from Lanolin | 1 pill at meal | 600IU or 15 mcg |
| Doctor Formulas Vitamin K2 200mcg | Menaquinone-7 | 1 pill at meal | 120mcg |
| Solgar Chelated Magnesium 400mg | glycinate chelate, oxide | 1 pill at meal | 420mg |
| Life Extension Vitamin C & Quercetin 1000mg | Ascorbic acid | 1 pill at breakfast + 1 pill at meal | 90mg |
| (optional) Vivomixx neo 9 | 1 sachet per day (fast for 1 hour before and after consuming) best working, stop eat for the day 18:00, take it 20:00 |
12/MAY - appointment to holistic, suggested take for ever a custom multivitamin (no branded)
| Ingredients / 2 caps: |
|---|
| Ester-C (Calcium Ascorbate) – 700 mg |
| Vitamin D3 – 7000 IU |
| Vitamin K2 VITAL – 140 µg |
| Magnesium (Bisglycinate Chelate) – 280 mg |
| Zinc (Zinc Bisglycinate) – 20 mg |
| B1 Thiamine – 70 mg |
| B2 Riboflavin Phosphate – 70 mg |
| B3 Nicotinamide – 70 mg |
| B5 Pantothenate – 70 mg |
| Biotin – 700 µg |
| Methyl B6 (Pyridoxal 5-Phosphate) – 70 mg |
| Methyl B12 – 700 µg |
| Folate Quatrefolic – 550 µg |
| Choline – 70 mg |
| Inositol – 70 mg |
| Copper Citrate – 1.5 mg |
| Chromium Picolinate – 70 µg |
| Selenium (L-selenomethionine) – 150 µg |
GPT:
The sensitivity of an individual's digestive system can vary widely due to several factors, and this can influence how they react to freshly cooked versus reheated food. Here are some key points to consider:
The sensitivity of an individual's digestive system can vary widely due to several factors, and this can influence how they react to freshly cooked versus reheated food. Here are some key points to consider:
1. Digestive Enzyme Activity
- Freshly Cooked Food: When food is freshly cooked, it may retain more moisture and heat, which can affect how enzymes in the digestive system break it down. Some people may find that their digestive enzymes are less effective at processing certain foods when they are hot and fresh.
- Reheated Food: Reheating food can change its texture and temperature, potentially making it easier for some individuals to digest. The cooling process may also allow for some breakdown of complex carbohydrates, making them easier to digest upon reheating.
2. Food Structure Changes
- Cooking alters the physical structure of food. For example, pressure cooking can break down cell walls and make food softer. However, this can also lead to a higher concentration of certain compounds that may be harder for some people to digest.
- When food is cooled and then reheated, the structure may change again, potentially making it easier for the body to process.